Table 4. Prevalence of putative virulence genes.
| Genea | No. of isolates (%) | Sourceb |
| Adhesins | ||
| esp | 1 (3) | C |
| hyl | 2 (5) | C |
| sgrA | 17 (44) | C, D |
| ecbA | 2 (5) | C, D |
| orf903 | 32 (82) | C, D, H |
| orf905 | 32 (82) | C, D, H |
| orf907 | 32 (82) | C, D, H |
| acm | 39 (100) | C, D, H |
| pilA | 35 (90) | C, D, H |
| pilB | 38 (97) | C, D, H |
| sagA | 39 (100) | C, D, H |
| Sugar metabolism | ||
| orf1481 | 19 (49) | C, D |
| IS-elements | ||
| IS16 | 31 (79) | C, D, H |
| IS256 in acm | 3 (8) | C, D, H |
| Community specific genes tentatively involved in sugar metabolism | ||
| EfmE4452_1561 | 28 (72) | C, D, H |
| EfmE4452_1565 | 28 (72) | C, D, H |
a
The genes acm, pilA, pilB and sagA are prevalent among all E. faecium strains; the genes EfmE4452_1561 and EfmE4452_1565 are uniquely present in community ARE isolates (this paper). All other genes are specifically enriched among hospital isolates [5], [7]–[8], [18]–[19], [52].
b
Source of the isolates carrying a particular gene: D = dog; C = cat; H = human.