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. 2011 Oct 22;40(4):1485–1498. doi: 10.1093/nar/gkr868

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© The Author(s) 2011. Published by Oxford University Press.

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 4. — Loss of Bu1a expression gene of rev1 cells correlates with loss of histone modifications associated with transcriptional activation. (A) Trimethylation of H3K4 at the BU1A promoter in WT, Bu1a-negative rev1 cells (Bu1a^lo) and rev1 cells enriched for expression of surface Bu1a (Bu1a^hi). (B) Acetylation of H3K9 and K14 at the BU1A promoter. (C) Acetylation of the H4 N-terminal tail at the BU1A promoter (D) Dimethylation of H3K9 at the BU1A (gray bars) and ρ-GLOBIN (white bars) promoters in WT, Bu1a-negative rev1 cells (Bu1a^lo) and rev1 cells enriched for expression of surface Bu1a (Bu1a^hi). Error bars represent standard error of the mean, P-values compared to WT (one-tailed, unpaired t-test): ns = not significant,*P < 0.075, **P < 0.01.