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. Author manuscript; available in PMC: 2012 Mar 1.
Published in final edited form as: Neurosci Lett. 1994 Feb 28;168(1-2):29–33. doi: 10.1016/0304-3940(94)90408-1

Fig. 3.

Fig. 3

The fimbria-evoked late hyperpolarization, revealed after bicuculline application, was blocked by the GABAB receptor antagonist 2-hydroxy saclofen. A–D: 3 responses to an identical fimbria stimulus are shown superimposed, triggered during current steps (top and bottom traces) or without any current injection (central trace). In control (A), a small IPSP was recorded. Following reduction of the GABAA IPSP (B), a late hyperpolarization was evoked after the residual GABAA IPSP. The residual GABAA IPSP is marked by an arrow, the late hyperpolarization is marked by an asterisk. The late hyperpolarization was blocked by 500 μM 2-hydroxy saclofen (C) reversibly (D). Stimulation occurred at the dot. Action potentials are truncated. Membrane potential, −71 mV; resting potential, −85 mV.