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. 2011 Nov 22;61(4):543–553. doi: 10.1136/gutjnl-2011-301012

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© 2012, Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions.

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Liver weight, inflammation, insulin and energy pathways during metabolic adaptation. (A) Liver weight; (B) TNFα, IL-6 and PAI-1 mRNA concentrations; (C–E) western blot analysis of phosphorylated and total proteins involved in (C) insulin signalling, (D) energy metabolism and (E) inflammation from mice fed a high-fat diet (HFD) which became diabetic (HFD-D), diabetes-resistant (HFD-DR) or mice fed a diet supplemented with gluco-oligosaccharides (HFD+GOS). Data are shown as mean ± SEM; *p<0.05, ***p<0.001 (unpaired Student t test; n=4 per group). IL, interleukin; PAI-1, plasminogen activator inhibitor 1; TNFα, tumour necrosis factor α.