. 2012 Mar;56(3):1342–1349. doi: 10.1128/AAC.05267-11

Table 2.

Distribution of the P. stuartii strains according to their epidemiological relationship, antimicrobial resistance phenotype, and IncA/C plasmid type

No. of strains	Pulsotype	ATB^a no.	Antimicrobial resistance phenotype of P. stuartii strains and their transconjugants^b												IncA/C PCR profile^c	Plasmid size (kb)^d
No. of strains	Pulsotype	ATB^a no.	AM	Cz	S	Sp	G	T	A	Su	Tp	C	Te	Cp	IncA/C PCR profile^c	Plasmid size (kb)^d
48^e	A	1	AM	Cz	S	Sp	G	T	A	Su	Tp	C	Te	—^g	1 2 3 4 5 6 - - 9 10 11 12	175
1	A	4	—	—	S	Sp	—	T	A	Su	Tp	C	Te	—	1 2 3 4 5 - - - - 10 11 12	100
7^f	B	2	AM	Cz	S	Sp	—	T	A	Su	Tp	C	Te	Cp	1 2 3 4 5 6 7 8 9 10 11 12	195
4	B	3	AM	Cz	S	Sp	G	T	A	Su	Tp	C	Te	Cp	1 2 3 4 5 6 7 8 9 10 11 12	195
4	C	3	AM	Cz	S	Sp	G	T	A	Su	Tp	C	Te	Cp	1 2 3 4 5 6 7 8 9 10 11 12	195

ATB, antibiotype.

AM, ampicillin; Cz, ceftazidime; S, streptomycin; Sp, spectinomycin; G, gentamicin; T, tobramycin; A, amikacin; Su, sulfonamides; Tp, trimethoprim; C, chloramphenicol; Te, tetracycline; Cp, ciprofloxacin.

According to Welch et al. (52).

Only the plasmids giving a positive hybridization with the repAIncA/C₂ probe are indicated.

Including five environmental strains.

Including one environmental strain.

—, sensitive phenotype except for ampicillin and gentamicin because the strains exhibited low-level resistance due to the basal expression of their chromosomal species-specific enzymes, AmpC (12) and AAC(2′)-I (16).