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. 2012 Mar 12;7(3):e33349. doi: 10.1371/journal.pone.0033349

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This is an open-access article, free of all copyright, and may be freely reproduced, distributed, transmitted, modified, built upon, or otherwise used by anyone for any lawful purpose. The work is made available under the Creative Commons CC0 public domain dedication.

This is an open-access article distributed under the terms of the Creative Commons Public Domain declaration, which stipulates that, once placed in the public domain, this work may be freely reproduced, distributed, transmitted, modified, built upon, or otherwise used by anyone for any lawful purpose.

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IL-12p40^−/− (n = 10), IL-12p35^−/− (n = 10), IFN-γR^−/− (n = 10) and C57Bl/6 (n = 5/experiment) (wild type, WT) mice were intranasally infected with 50 CFU/25 µl SchuS4. Beginning on day 3 after infection, mice were treated once daily with 5 mg/kg LVF diluted in 5% dextrose water for 14 days. * = p<0.05 compared to untreated mice and LVF treated IL-12p40^−/−, IL-12p35^−/−, IFN-γR^−/− animals. ** = p<0.05 compared to all other groups of mice. Data in each graph is representative of 2 experiments of similar design.