Fig 1.
Effects of single null mutations of rsbR genes on the YtvA-dependent light activation of the GSR. All strains contained a copy of lacZ, controlled by the σB-dependent ctc promoter, and pYtvA, a plasmid for overexpression of ytvA. The β-galactosidase activity was determined 2 h after IPTG induction of YtvA overexpression. Absolute activation expressed in Miller units was normalized on the activation of the “wild-type” strain PB198/pYtvA in the light, which was set to 1. Filled bars correspond to dark conditions; open bars correspond to light conditions. The other strains used were PB491/pYtvA (ΔrsbRA), PB528/pYtvA (ΔrsbRB), PB574/pYtvA (ΔrsbRC), and PB520/pYtvA (ΔrsbRD).