Fig 4.

Effect of ADAR1 or PKR deficiency on MV-induced activation of ATF2. CON^kd, PKR^kd, and ADAR1^kd cells were either left uninfected (UI) or infected with WT, V^ko, or C^ko MV, as indicated. At 24 h postinfection, whole-cell extracts were prepared and analyzed by Western immunoblotting with antibodies against ATF2, phospho-Thr71-ATF2, and α-tubulin.