Table 2.
Cardiac tissue omega-3 polyunsaturated fatty acid content.
| EPA | DHA | Omega-3 index | |
|---|---|---|---|
| RIGHT ATRIUM | |||
| Placebo | |||
| VF− | 0.11 ± 0.08 | 0.16 ± 0.10 | 0.27 ± 0.18 (n = 3) |
| VF+ | 0.36 ± 0.12 | 0.58 ± 0.17 | 0.91 ± 0.25 (n = 6) |
| n−3 PUFA | |||
| VF− | 2.04 ± 0.31* | 2.89 ± 0.33* | 4.92 ± 0.59* (n = 23) |
| VF+ | 1.62 ± 0.25* | 2.80 ± 0.37* | 4.41 ± 0.61* (n = 22) |
| LEFT VENTRICLE | |||
| Placebo | |||
| VF− | 0.12 ± 0.06 | 0.19 ± 0.06 | 0.31 ± 0.11 (n = 3) |
| VF+ | 0.34 ± 0.05 | 0.45 ± 0.10 | 0.80 ± 0.13 (n = 6) |
| n−3 PUFA | |||
| VF− | 2.98 ± 0.30* | 3.00 ± 0.17* | 5.98 ± 0.43* (n = 23) |
| VF+ | 2.27 ± 0.28* | 2.80 ± 0.20* | 4.41 ± 0.43* (n = 21) |
All values are expressed of % of the total lipid content. EPA, eicosapentaenoic acid; DHA, docosahexaenoic acid; omega-3 index, EPA + DHA, *P < 0.01 placebo vs. n−3 PUFA (omega-3 polyunsaturated fatty acids). There were no significant differences between VF− (resistant to ventricular fibrillation) and VF+ (susceptible to ventricular fibrillation) for either the placebo or n−3 PUFA treated animals.