Table 1. Patients' characteristics at enrolment of the study.
ATG treatment* | Sex † | Age [years] | CRP [mg/dl]♣ | Leukocytes [G/l]♦ | Hb [g/l] | concommitant immune-suppressive therapy‡ | TX-organ | treated condition |
N | M | 56 | 0.7 | 9.2 | 158 | CsA, MMF, GC, aIL2R | L | chronic emphysema |
N | M | 57 | 2.3 | 5.0 | 108 | CsA, GC, MMF | K | chronic GN |
N | M | 46 | 0.7 | 4.6 | 126 | aIL2R, FK-506, GC | L | CirrhHep |
N | M | 43 | 0.7 | 8.3 | 117 | aIL2R, FK-506, GC | K | chronic GN |
N | M | 61 | 0.7 | 2.9 | 100 | aIL2R, FK-506, GC | L | CirrhHep |
N | F | 50 | 3.5 | 4.8 | 100 | aIL2R, RAP, MMF, GC | K | PN |
N | M | 44 | 0.7 | 4.4 | 162 | CsA, GC, MMF | K | Alport syndrome |
N | M | 45 | 0.7 | 10.6 | 136 | FK-506, GC, MMF | K | chronic graft rejection |
N | F | 59 | 0.4 | 9.0 | 129 | aIL2R, FK-506, MMF,GC | K | PN |
N | M | 44 | 0.3 | 9.3 | 129 | FK-506, MMF, GC | K | IgA nephropathy |
N | M | 51 | 0.3 | 3.8 | 142 | FK-778, FK-506, GC | L | CirrhHep |
Y | M | 51 | 0.7 | 4.7 | 133 | ATG, FK-506, RAP, GC | K, P | DM I |
Y | F | 48 | 0.7 | 6.6 | 108 | ATG, FK-506, RAP, GC | K, P | DM I |
Y | F | 29 | 0.3 | 8.0 | 105 | ATG, FK-506, MMF, GC | K, P | DM I |
Y | F | 24 | 0.4 | 7.9 | 112 | ATG FK-506, RAP, GC | K, P | DM I |
Y | M | 41 | 0.5 | 7.1 | 155 | ATG, FK-506, MMF, GC | K, P | DM II |
no, N; yes, Y;
male, M; female, F;
C-reactive protein ,CRP (normal values: 0–0.6 mg/dl);
normal values: 3.8–10.5 G/l; hemoglobin, Hb (normal values: 120–157 g/l).
cyclosporine A, CsA; mycophenolate mofetil, MMF; glucocorticoids, GC; monoclonal anti-human IL-2 receptor antibodies (daclizumab, zenapax™), aIL2R; tacrolimus, FK-506; rapamycine, RAP; monoclonal anti-human CD52 antibodies (alemtuzumab, campath™), anti-CD52 Ab; leflunomide analogue FKK-778, FKK-778; ATG Fresenius, ATG.
Decompensated cirrhosis hepatis, CirrhHep; diabetes mellitus with nephropathy, DM; glomerulonephritis, GN; Kidney, K; lung, L; pancreas, P; polycystic nephropathy, PC; transplant, TX.