Table 1. Subject scores for the parkinsonism-related metabolic patterns in normal and MPTP-lesioned monkeys.

	Cohort A		Cohort B
	Control	MPTP	Control	MPTP
Hemispheric analysisa
PRP1 (42.9)	0.00±0.07	1.73±0.21	0.12±0.07	0.83±0.07
PRP2 (43.3)	0.09±0.07	1.67±0.23	−0.04±0.06	0.81±0.07
PRP3 (27.8)	0.42±0.12	2.05±0.29	0.00±0.16	1.77±0.12
PRP4 (27.2)	0.00±0.19	2.10±0.29	0.38± 0.16	1.68±0.15
Whole-brain analysisb
PRP1	0.00±0.09	1.73±0.31	0.12±0.09	0.83±0.08
PRP2	0.09±0.09	1.67±0.33	−0.04±0.09	0.81±0.08
PRP3	0.42±0.16	2.05±0.42	0.00±0.20	1.77±0.14
PRP4	0.00±0.22	2.10±0.41	0.38± 0.15	1.71±0.16
PRP5c (48.2)	0.00±0.09	1.70±0.31	0.13±0.09	0.78±0.04

MPTP, 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine; PRP, parkinsonism-related pattern.

Subject scores were presented as mean±standard error. Bold indicated the scans used to identify each of the significant PRP networks. In each cohort, subject scores for these networks were elevated in the parkinsonian animals compared with the controls. The eigenvalue associated with each PRP derivation is given in parentheses as the percent of the subject × voxel variance accounted for (Spetsieris and Eidelberg, 2011).

^aSubject scores for PRPs generated by hemispheric analysis (see text).

^bSubject scores for the whole brain computed as the left/right average of subject scores for PRPs generated on a hemispheric basis.

^cSubject scores for PRP5 were computed as part of a whole-brain spatial covariance analysis.