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. 2011 Feb 25;60(6):781–792. doi: 10.1007/s00262-011-0987-5

Table 1.

Prostate cancer patients have preexisting T cells specific for AR LBD-derived peptides

Patient Peptide-specific T-cell responses
AR677 AR700 AR708 AR742 AR761 AR805 AR811 AR814 AR859 AR862 Influenza
D04 + + +* + +
D05 + ns
P01 + +* +* + +
P03 ns ns ns ns + ns ns ns
P07 + + +* +* + + +
P08 + + + + + + + +
P09 + + + + ns
P11 + +
P12 + + + + +
P16 + +
P17 + + +
P19 ns ns ns ns + + ns ns ns
P20 ns ns ns ns ns + ns ns
P21 ns ns ns ns + + + ns ns ns
P24 ns ns ns ns + ns + ns ns
Frequency of patients with T-cell responses 4/10 (40%) 3/10 (30%) 2/10 (20%) 1/10 (10%) 6/15 (40%) 8/15 (53%) 8/15 (53%) 2/10 (20%) 5/15 (33%) 4/10 (40%) 8/8 (100%)

AR LBD-derived peptides or an influenza matrix protein positive control were used to culture peptide-specific T cells from as many as fifteen HLA-A2+ patients with prostate cancer (listed in the first column). After 2–8 weekly in vitro peptide stimulations, T-cell cultures raised against individual peptides were tested for the presence of peptide-specific T cells and prostate cancer cell lysis using cytotoxicity assays. Patients from whom peptide-specific CTL could be detected after 2–8 peptide stimulations are indicated with a ‘+’, whereas patients from whom CTL could not be detected are indicated with a ‘−’. Peptides that were not sampled for certain patients are indicated by ‘ns’. Peptide-specific T-cell cultures that were found to lyse prostate cancer cells are indicated by a ‘*’. The frequency of HLA-A2+ prostate cancer patients that recognize each particular peptide is listed in the bottom row