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. Author manuscript; available in PMC: 2013 Apr 1.
Published in final edited form as: Gend Med. 2012 Mar 22;9(2):103–111. doi: 10.1016/j.genm.2012.02.003

Figure 3. Inhibition of androgen receptors or NO reduces testosterone-induced vasorelaxation.

Figure 3

Figure 3

A. After the Af-Art were treated with the androgen receptor antagonist, flutamide (10−5mol/L), testosterone (TES)-induced relaxation of the Af-Art was blocked (n=9). After L-NAME (3×10−4mol/L) was added in the bath, the diameter of Af-Art was decreased from 11.3±1.1 µm to 8.6±0.3 µm. Then testosterone (TES) was added into bath in the presence of L-NAME, the diameter of Af-Art was dilated to 10.0±0.4µm (*p<0.05, v.s. L-NAME; n=8). B. The dilatory effect expressed in percentage. Compared with norepinephrine (NE), in the presence of L-NAME, TES’s dilatory effect was significantly blunted (p<0.05 v.s. TES).