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. 2012 Apr 11;7(4):e33787. doi: 10.1371/journal.pone.0033787

Table 2. The most generally down-regulated genes in atherosclerotic plaques analysed with TaqMan Low Density array.

Gene abbreviation Gene ID Average FC
ITLN1 (intelectin 1) 55600 −568.2 (p<0.001)
APOD (apolipoprotein D) 347 −103.9 (p<0.001)
DUSP26 (dual specificity phosphatase 26) 78986 −21.3 (p<0.001)
CASQ2 (calsequestrin 2) 845 −23.6 (p<0.001)
RGS5 (regulator of G-protein signaling 5) 8490 −13.0 (p<0.001)
TCEAL2 (transcription elongation factor A (SII)-like 2) 140597 −18.6 (p<0.001)
DES (desmin) 1674 −15.8 (p=0.025)
CALD1 (caldesmon 1) 800 −10.4 (p<0.001)
PPP1R3C (protein phosphatase 1) regulatory (inhibitor) subunit 3C) 5507 −9.5 (p<0.001)
ADRA2C (adrenergic, alpha-2C-, receptor) 152 −12.9 (p<0.001)
CSRP2 (cysteine and glycine-rich protein 2) 1466 −8.6 (p<0.001)
SPEG (SPEG complex locus) 10290 −4.2 (p<0.001)
CNN1 (calponin 1) 1264 −4.3 (p<0.001)
C6orf117 (chromosome 6 open reading frame 117) 112609 −8.7 (p<0.001)
LMOD1 (leiomodin 1) 25802 −6.5 (p<0.001)
RAMP1 (receptor activity modifying protein 1) 10267 −5.4 (p<0.001)

The results are shown as an average fold change (FC) compared to control arteries.