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. 2012 Apr 15;23(8):1446–1456. doi: 10.1091/mbc.E11-09-0797

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© 2012 Wang et al. This article is distributed by The American Society for Cell Biology under license from the author(s). Two months after publication it is available to the public under an Attribution–Noncommercial–Share Alike 3.0 Unported Creative Commons License (http://creativecommons.org/licenses/by-nc-sa/3.0).

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FIGURE 8: — Growth and reproductive phenotypes of NSN1^OX plants. (A–D) Adult plants of NSN1^OX were dwarf and bushy (A) and produced mostly short siliques (arrowhead, B). They lost apical dominance in the main shoot (arrowhead, C) and developed a terminal flower (arrow, C) consisting of deformed sepals, petals, and stamens (arrow, D), as well as unfused carpels with ectopic ovules (arrowhead, D). (E–G) NSN1^OX plants produced flowers with an increased number of petals (E) and short and curled siliques (F) containing unfertilized ovules (arrowhead, G) and aborted embryos (arrow, G). Siliques developed from a gynoecium of three fused carpels were often seen in NSN1^OX as in ag/+ mutant (arrows, F). Scale bars, 70 mm (A), 10 mm (B), 20 mm (C), 2 mm (D–G). (H) A proposed role of NSN1 as a general negative regulator of AG in the control of inflorescence meristem (IM) determinacy and floral organ identity in Arabidopsis. It may exert its role in a pathway independent of AP2.