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. Author manuscript; available in PMC: 2012 Oct 1.
Published in final edited form as: Neurotoxicology. 2011 Jul 23;32(5):567–577. doi: 10.1016/j.neuro.2011.06.009

Fig. 2. Dieldrin induces phosphorylation of PKCδ-tyr311 in dopaminergic neuronal cell model.

Fig. 2

N27 dopaminergic cells treated with dieldrin (60μM) were lysed and ~50μg of protein was used to determine tyrosine phosphorylation of PKCδ by Western blot (A) Dieldrin-treated cells depicted increases in PKCδ-tyr311 phosphorylation as compared to the untreated cells and proteolytic cleavage of PKCδ native protein at 60 min. (B) Densitometric analysis of the immunoblots are expressed as relative band intensities of phospho-PKCδ-tyr311 to native-PKCδ (mean ± S.E.M. of three independent experiments). **p < 0.01.