Table 6.

Seven athletes with a positive diagnosis of a disease associated with sudden cardiac death

Athlete	Age (years)	Ethnicity	Sport	FH	Symptoms	Abnormal ECG	Deep T-wave inversions	Echo result	CMR result	Diagnosis
1	29	West-Asian	Athletics	None	None	WPW – sustained during exercise	–	Normal	–	WPW – successful ablation
2	13	Black	Football	None	None	WPW – sustained during exercise	–	Normal	–	WPW
3	13	West-Asian	Tennis	None	Difficulty training at high heart rates and systolic murmur	RVH and LVH voltage criteria	None	IVS 14 mm, PWT 13 mm, LVIDd 41 mm, subaortic septal hypertrophy (18 mm), mitral valve prolapse and diastolic dysfunction	Not done – patient left Qatar	HCM
4	18	Black	Basketball	SCD of mother (<35 years)	Chest pains on exertion	ARVC	V1–V6	ARVC	Confirmed ARVC	ARVC
5	30	West-Asian	Football	SCD of uncle (<35 years)	Impact syncope during exercise	RVH and LVH voltage criteria	V3–V6	IVS 13 mm, PWT 16 mm, LVIDd 40 mm, abnormal LV wall motion and diastolic dysfunction	Not done – patient left Qatar	HCM
6	22	Black	Basketball	None	Post screening: two episodes of sustained VT during exercise requiring cardioversion	LVH voltage criteria, complete LBBB and RAD	V3–V6	IVS 14 mm, PWT 12 mm, LVIDd 58 mm and abnormal LV wall motion	Normal CMR – echo dimensions confirmed – no LGE/oedema or systolic dysfunction	HCM
7	18	West-Asian	Football	None – sister QTc <460 ms	None	Serial ECGs – long QT (500 ms)	–	Normal	–	Long QT syndrome

ARVC, arrhythmogenic right ventricular cardiomyopathy; CMR, cardiac magnetic resonance; Echo, echocardiography; FH, family history; HCM, hypertrophic cardiomyopathy; IVS, interventricular septum; LBBB, left bundle branch block; LGE, late gadolinium enhancement; LVH, left ventricular hypertrophy; LVIDd, left ventricular internal diameter during diastole; PWT, posterior wall thickness; RAD, right axis deviation; RVH, right ventricular hypertrophy; SCD, sudden cardiac death; VT, ventricular tachycardia; WPW, Wolff–Parkinson–White.