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Summary model of nongenomic steroid [TES and 17β-estradiol (E2)] signal transduction in CASM. ER, estrogen receptor; AR, putative androgen receptor; PI3, phosphoinositide 3-kinase; Akt, protein kinase B; sGC, soluble guanylyl cyclase; BK, BKCa channel. Broken line indicates that TES may also stimulate production of NO from endothelial nitric oxide synthase (44).
