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. 2012 May;191(1):145–162. doi: 10.1534/genetics.111.137190

Table 2 . Smaller deficiencies and individual genes, which confirmed original interactions.

DrosDel deficiency Subdividing deficiencies Genes identified sev-GAL4, UAS-dgo interaction sev-GAL4, UAS-pk
interaction
Df(1)ED6957 Nonea ND
Df(1)ED7161 Df(1)ED7153 CG17788 (no RNAi), CG15926 (no RNAi), CG2556, CG15730, CG11146, CG32653 CG15730: enh(rota) Su (chir)b
Df(1)Exel6244 CG11146: nob Enh (rota + chir)
Df(1)ED447 Nonec CG6461, CG6470, CG6335, CG10548, CG6481, CG15042, CG15047
Df(2L)ED62 Df(2L)Exel8003 ds, CG2863
Df(2L)ED94
Df(2L)ED49
dsUA071
Df(2L)ED623 Df(2L)ED611 CG13388, CG13399, CG13400 Su (chir)b Su (rota + chir)b
Df(2L)ED647 Enh (rota)
Df(2L)Exel8021
Df(2L)ED793 Df(2L)b87a25 CG15283, CG4491, noc, CG15284, CG3474 Enh (rota) Nob
Df(2L)Exel6036
Df(2L)Exel6035
Df(2L)Exel8033
Df(2L)ED1315 Nonec CG31683, CG31687, CG18858 Enh (rota) Enh (rota + chir)
Df(2R)ED1618 Df(2R)ED1673 Pk
Df(2R)ED1673 Df(2R)ED1618 Pk
Df(3L)ED201 Df(3L)Exel6084 CG7004 Enh (rota) Enh (rota + chir)b
fwd Df(3L)Exel9057
fwdneo1
Df(3L)ED207 Nonea ND
Df(3L)ED220 Df(3L)st[b11]d ND
Df(3L)ED230 Df(3L)Al29 CG11438, CG11426 ND ND
Df(3L)AK1
Df(3L)ED5017 Nonee ND
Df(3R)ED7665 Nonec CG14612 (no RNAi), CG1070 (no RNAi), CG1019, CG10098 (ND), CG10068 Enh (rota)f No
Df(3R)ED5577 Df(3R)Exel7316 ND
Df(3R)ED5623 Nonea ND
Df(3R)ED5644 Df(3R)Exel6267 ND
Df(3R)ED5942 Df(3R)Cha9 Dl
Df(3R)Dl[RF]
Df(3R)Dl[7]
Df(3R)ED10639 Df(3R)Exel7329 CG6889, CG6815, CG6814, CG6864, CG12785, CG6963, CG31283 Enh (rota) ND

The DrosDel and smaller deficiencies are listed in the first and second columns, respectively. The third column lists all genes that were tested by RNAi knockdown for PCP phenotypes. Genes that affected PCP when knocked down (compare Table 3) and reproduced the effects of the original deficiency are indicated in boldface type. Df(2L)ED62 and Df(3R)ED5642 covered ds and Dl, respectively (compare Figure S1). Columns 4 and 5 list genes and the quality of their genetic interactions that reproduced the original genetic interactions with screen genotypes.

a

Full coverage by other dfs.

b

Genetic interaction is different from what was observed with initial deficiency.

c

Genes tested based on exclusion from smaller Dfs.

d

Approximately 60 gene overlap.

e

No smaller Dfs, close to centromere.

f

Genetic interaction is a subset of what was observed originally.