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. 2011 Dec 22;5(3):351–365. doi: 10.1242/dmm.002873

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© 2012. Published by The Company of Biologists Ltd

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial Share Alike License (http://creativecommons.org/licenses/by-nc-sa/3.0), which permits unrestricted non-commercial use, distribution and reproduction in any medium provided that the original work is properly cited and all further distributions of the work or adaptation are subject to the same Creative Commons License terms

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Fig. 7. — Conditional deletion of Sall1 in dorsal PCs indicates that Sall1 regulates PC fate progression. Sall1 (red) expression is localized to the ventral telencephalon and does not extend into the dorsal cortex in Sall1-cKO^Emx1 animals (arrow, B), unlike controls (A) at E12.5. Tuj1 staining (green) of cortical neurons at E12.5 identified an increase in preplate cell number in Sall1-cKO^Emx1 (D) animals compared with controls (C). CSPG (red) immunostaining of the subplate and marginal zone identified an increase in these structures in Sall1-cKO^Emx1 (F) animals compared with controls (E) at E18.5. Tbr2 (red) immunostaining at E18.5 identified an increase in IPCs in Sall1-cKO^Emx1 (H) animals compared with controls (G). PC, progenitor population; PP, preplate; SP, subplate; CP, cortical plate; MZ, marginal zone. Scale bar: 100 μm (C,D,G,H); 350 μm (E,F); 400 μm (A,B).