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. 2012 Apr 19;13(4):4968–4981. doi: 10.3390/ijms13044968

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© 2012 by the authors; licensee Molecular Diversity Preservation International, Basel, Switzerland.

This article is an open-access article distributed under the terms and conditions of the Creative Commons Attribution license (http://creativecommons.org/licenses/by/3.0/).

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Cerebral infarction after PMCAO in vehicle- or DAEE-treated mice. (A) 2,3,5-triphenyltetrazolium chloride (TTC) staining of coronal brain sections (2-mm thick) obtained 96 h after PMCAO from the vehicle-control mice; (B) TTC staining of coronal brain sections (2-mm thick) obtained 96 h after PMCAO from the DAEE–treated mice; (C) Brain infarct area measured 96 h after PMCAO. Brains were removed, and the forebrains were sliced into five coronal 2-mm sections. The section numbers correspond to the numbers of “A” and “B”. The brain infarct area was not significantly different between the two groups (vs. vehicle; Student’s t-test; n = 6 or 5). The values are expressed as the means ± SEs; (D) Brain infarct volume measured 96 h after PMCAO. There was no significant difference in infarct volume between both groups (Student’s t-test; n = 6 or 5). The values are expressed as the means ± SEs.