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. 2012 May 7;7(5):e36654. doi: 10.1371/journal.pone.0036654

Figure 2. Histological causes of post-transplant proteinuria.

Figure 2

Figures shows histological causes of proteinuria in all patients (1A), urine protein <1 g/d (1B), between 1–3.5 g/d (1C), and over 3.5 g/d (1D). TG was the leading cause of overall post-transplant proteinuria and each subgroup, followed by IgAN, chronic rejection (TG excluded), and acute rejection. For proteinuria below 1.0 g/d, acute rejection was the second most important cause of proteinuria, accounting for 25% in this subgroup; only one (3%) patient was diagnosed as having IgAN. For proteinuria between 1–3.5 g/d, the incidence of acute rejection decreased to 4.9%, while the incidence of IgAN increased to 24.4%. In proteinuria over 3.5 g/d, IgAN accounted for 19%, and no patient was diagnosed as having acute rejection. TG, transplant glomerulopathy; IgAN, IgA nephropathy; AR, acute rejection; CR, chronic rejection; TA/IF, tubular atrophy and interstitial fibrosis; FSGS, Focal segmental glomerulosclerosis; MPGN, Membranoproliferative glomerulonephritis; HSPN, Henoch-Schönlein purpura nephritis; DN, diabetic nephropathy; MSP, mesangial proliferative glomerulonephritis; MN, Membranous nephropathy.