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. 2012 May;86(9):5264–5277. doi: 10.1128/JVI.06913-11

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Fig 9 — (A) Immunoblots of electrophoretically separated virions of wild-type HSV-1(F) (lane 1), YK453 (VP22LL235AA) (lane 2), YK454 (VP22LL235AA-repair) (lane 3), YK456 (VP22LL251AA-repair) (lane 4), YK455 (VP22LL251AA) (lane 5), and YK476 (ΔUL41) (lane 6) purified by sucrose gradient centrifugation and blotted with an antibody to VP22. (B) Immunoblots of electrophoretically separated virions of wild-type HSV-1(F) (lane 1), YK453 (VP22LL235AA) (lane 2), YK454 (VP22LL235AA-repair) (lane 3), YK455 (VP22LL251AA) (lane 4), YK456 (VP22LL251AA-repair) (lane 5), and YK476 (ΔUL41) (lane 6) purified by sucrose gradient centrifugation and blotted with an antibody to VP5 or VP16.