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. 2012 Apr 23;109(19):7523–7528. doi: 10.1073/pnas.1204391109

Fig. 1.

Fig. 1.

Characterization of BSA–CNH complexes. (A) Schematic illustration of the BSA–CNH complex. (B) UV–vis–NIR spectrum of BSA–CNH in PBS buffer. (Inset) Photograph of BSA–CNH complex dispersions in (1) DMEM, (2) PBS, and (3) artificial seawater. (C) Temperature changes in BSA–CNH complex dispersions under continuous 670-nm laser irradiation at various power levels: 50 mW (∼3 mW/mm2), 100 mW (∼5 mW/mm2), 150 mW (∼8 mW/mm2), 200 mW (∼10 mW/mm2), 250 mW (∼13 mW/mm2), and 300 mW (∼15 mW/mm2). (D) Temperature changes in various nanocarbon complexes under continuous 785-nm laser irradiation at 300 mW (∼24 mW/mm2) for 5 min. Hipco, high-pressure carbon monoxide; CoMoCAT, cobalt–molybdenum catalyst. (E) Cytotoxicity tests of various BSA–functionalized nanocarbon complexes in NIH 3T3, Colon-26, and C6 cells. The standardized percentage of mitochondrial enzyme activity compared with control. Statistical analyses were performed using ANOVA (Tukey’s test). aP < 0.0005; bP < 0.0001; cP < 0.025.