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. Author manuscript; available in PMC: 2013 Mar 1.
Published in final edited form as: Pain. 2012 Jan 31;153(3):710–721. doi: 10.1016/j.pain.2011.12.013

Figure 2.

Figure 2

Upregulation of pCaMKII is evident at 35 days after contusive SCI. (A) Western blot data from the same naïve, sham and SCI rats indicated that SCI rats had significantly greater levels of activated pCaMKII than the other groups (p < 0.0001). (B) No differences existed between the groups in terms of nonphosphorylated CaMKII expression (p > 0.05). (C) Representative Western blots for CaMKII (middle) and pCaMKII (top). GAPDH (bottom) is included as the loading control.