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. Author manuscript; available in PMC: 2013 Jun 15.
Published in final edited form as: J Immunol. 2012 May 16;188(12):5859–5866. doi: 10.4049/jimmunol.1102328

Figure 4. Absent IL-7 signaling results in peripheral T lymphopenia in K7 mice.

Figure 4

(A) Spleen cell analysis of K7 mice. Total splenocytes from WT and K7 mice were assessed for TCRβ and CD4/CD8 expression. Data are representative of ten independent experiments.

(B) Peripheral T lymphopenia in K7 mice. LN T cell numbers were quantified in WT, IL7KO and K7 mice. Data are the summary from ten independent experiments.

(C) Increased CD4/CD8 ratio in K7 LN T cells. CD4 versus CD8 ratio of LN T cells were determined in WT and K7 mice. Data are summary of ten independent experiments.

(D) Reduced surface CD8 levels on K7 CD8 LN T cells. Surface CD8α expression was determined on WT and K7 LN T cells. Data are representative of ten independent experiments,

(E) K7 LN T cells express low levels of Bcl-2. Intracellular Bcl-2 expression was determined in WT and K7 CD8 LN T cells. Data are the summary of three independent experiments.

(F) IL-7Rα expression on CD4 and CD8 T cells of WT and K7 mice. Relative expression of surface IL-7Rα expression was assessed by mean fluorescence intensities on WT and K7 T cells. Data are the summary of three independent experiments.

(G) Peripheral IL-7-deficiency results in significantly decreased memory phenotype CD8 T cells. LN CD8 T cells were stained for CD122 and CD44 expression (left). Percentages of memory phenotype-like cells were determined in multiple experiments (right). Data show the result of ten independent experiments.