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. Author manuscript; available in PMC: 2013 Jun 15.
Published in final edited form as: Biol Psychiatry. 2012 Apr 27;71(12):1090–1098. doi: 10.1016/j.biopsych.2012.03.013

Figure 1.

Figure 1

FGFRcKO mice lacked fgfr2 early in development and had persistent neuroanatomical deficit. A,B: Images of fgfr2 IIIc TM mRNA localization by in situ hybridization in the hippocampus at P0 showing decreased expression in FGFR2cKO mice. C,D: Cresyl violet staining showing decreased volume of hippocampus in a FGFR2cKO compared to a control mouse. Scale bar=200 µm.