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. 2006 Feb 1;6(4):1–57.

Table 6: Metal-on-Metal Hip Resurfacing Arthroplasty: Clinical Outcomes of Case Series*.

Study Study Period Mean Duration of Follow-up, Years Number of Patients (Hips) Mean Age, Years (Range) Preoperative Diagnosis Number (%) Prosthesis Number of Patients Available for Follow-up
De Smet et al. 2005 (36) 1998–2004 2.8 (2–5.0) 252 (268) 49.7 (16–75) OA: 203 (80.6)
RA: 9 (3.57)
Necrosis: 22 (7.26)
Congenital dislocation: 12 (4.76)
Traumatic: 3 (1.19)
Neurometabolic: 1 (0.4)
Other: 2 (0.79)
BHR All except 3 patients who died
Back et al. 2005 (37) 1999–2001 3 (2–4.4) 230 52.1 (18–82) OA: 203 (88.3)
RA: 3 (1.3)
Avascular necrosis: 12 (5.2)
Neurometabolic: 2 (0.9)
Other: 10 (4.3)
BHR All
Treacy et al. 2005 (38) 1997–1998 130 (144) 52.1 (17–76) OA: 125 (87)
RA: 2 (1)
Avascular necrosis: 10 (7)
Developmental dysplasia: 3 (2)
Other: 4 (3)
BHR At 5 years: 107 (76) hips
Lilikakis et al. 2005 (39) 2002–2002 2.8 (2–3.2) 66 (70) 51.5 (23.3–72.7) OA: 64 (97)
Osteonecrosis: 1 (1)
Chondrolysis: 1 (1)
Cormet 2000: femoral and acetabular components were coated with hydroxyapatite 59/66 radiographs were available

60/66 patients returned the questionnaire at the 2-year follow-up
Amstutz et al. 2004 (34) 1996–2000 3.5 (2.26.2) 355 (400 hips) 48.2 (1577) OA: 262 (66)
Osteonecrosis: 36 (9)
Developmental dysplasia: 43 (10.8)
Posttraumatic c arthritis: 31 (7.8)
Leg-Calve-Perthes disease: 10 (2.5)
Slipped capital femoral epiphysis: 7 (1.8)
Ankylosing spondylitis: 4 (1)
Juvenile rheumatoid arthritis: 3 (0.8)
Rheumatoid arthritis: 3 (0.8)
Melorheostosis: 1 (0.3)
Previous operations: 25 (6.3)
Conserve Plus 352
Daniel et al. 2004 (35) 1994–2001 (but not 1996)* 3.3 (1.1-8.2) 384 (446 hips) 48.3 (26.8-54.9) Primary OA McMinn (43 hips, 1994–1995) 43
prostheses BHR (403 hips 1997-2001) 403
prostheses
All
Beaule et al. 2004 (40) NR 3 (2–5) 83 (94) 34.2 (15–40) OA: (24.4)
Trauma: (18.1)
Osteonecrosis: (18.1)
Developmental dysplasia of the hip: (19.1)
Rheumatoid diseases: (6.4)
Slipped capital femoral epiphysis: (4.3)
Leg-Calve-Perthes disease: (6.4)
Ankylosing spondylitis: (3.2)
Conserve Plus 81
Beaule et al. 2004 (41) 1993–1996 8.7 (7.2–10) 39 (42 hips) 47.5 (22–69) OA: 23 (55)
Osteonecrosis: 7 (16.7)
Hip dysplasia: 4 (9.5)
Arthrokatadysis: 3 (7)
RA: 2 (4.8)
Slipped capital femoral epiphysis: 2 (4.8)
Legg-Calve-Perthes: 1 (2.4)
McMinn
femoral components:
All were cemented.

acetabular component:
Was cemented into the native acetabulum in 19 hips. 16 hips were reconstructed by cementing the McMinn acetabular component into the cemented into a cementless cobalt chromium alloy, porous-coated surface replacement socket due to absence of a locking mechanism. 7 hips were hydroxyapatite coated.
All
*

186 patients operated on in 1996 were excluded from the study because a unique pattern of failure, (believed to be due to the manufacture) occurred in the implants used; OA indicates osteoarthritis; RA, rheumatoid arthritis; AN, avascular necrosis; BHR, Birmingham Hip Resurfacing; NR, not reported.