FIG 5. Proposed mechanisms of Imidazoquinoline TLR8 agonist activation of human newborn monocytes and DCs via adenosine-refractory and caspase-1-dependent pathways.
3M-002 agonists are refractory to inhibitory effects of the adenosine/cAMP pathway, which skews neonatal immunity against Th1-polarizition (A), can overcome impaired IL-1β responses of neonatal DCs to LPS/ATP (B) independently of exogenous ATP (C).