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. Author manuscript; available in PMC: 2013 Aug 1.
Published in final edited form as: Cell Microbiol. 2012 Apr 16;14(8):1231–1241. doi: 10.1111/j.1462-5822.2012.01793.x

Figure 3. ETEC induces LT-dependent MAPK activation.

Figure 3

A. Immunoblotting of total and phosphorylated ERK1/2, p38, and JNK after treating HCT-8 cells with TNF-α (20 ng/ml, 30′) or infecting them with either wt, ΔeltA, or ΔeltA/pLT ETEC for 2 h. B. Quantification of relative phosphoprotein abundance from data shown in panel A. Data are presented as mean ± SEM, n = 3–4, with asterisks (*) used to indicate a statistically significant difference from wt ETEC (p < 0.05, Student’s t test). C. Immunoblotting of total and phosphorylated ERK1/2, p38, and JNK after treating HCT-8 cells with TNF-α (20 ng/ml, 30′) or infecting them with variable amounts of either wt or ΔeltA ETEC (MOIs ranging from 25–100).