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. Author manuscript; available in PMC: 2013 Jul 30.
Published in final edited form as: J Neurosci Methods. 2012 Jun 16;209(1):127–133. doi: 10.1016/j.jneumeth.2012.06.006

Figure 4.

Figure 4

High K+ aCSF stimulated OXT (F(6,5) = 11.76, p < 0.0001) and AVP (F(6,5) = 6.90, p = 0.002) release (A). Black bar represents the time in which high K+ was in aCSF. Systemic injection of hypertonic saline (black arrow) caused a transient increase in OXT (F(12,5) = 7.33, p < 0.0001) while causing a delayed and long lasting increase in AVP (B) (F(12,5) = 8.65, p < 0.0001). RM ANOVA and a post-hoc Tukey test were performed to compare basal levels against post drug levels. All in vivo microdialysis data are expressed as percent of baseline ± SEM. *p < 0.05, **p < 0.01, ***p < 0.001 compared to basal levels in the PVN.