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. 2012 Jul;166(6):1804–1814. doi: 10.1111/j.1476-5381.2012.01881.x

Table 2.

Pharmacological profiles of [3H]-NMS and [3H]-CGP12,177 binding sites in human bronchus and lung

[3H]-NMS binding sites [3H]-CGP-12,177 binding sites
Pirenzepine (M1- subtype) AF-DX 116 (M2- subtype) Darifenacin (M3- subtype) ICI-89,406 (β1- subtype) ICI-118,551 (β2- subtype)
Region of the airway pKihigh (%) pKilow pKihigh (%) pKilow pKihigh (%) pKilow pKihigh (%) pKilow pKihigh (%) pKilow
Segmental bronchus 7.4 ± 0.2 7.3 ± 0.1 (44 ± 6%) 5.8 ± 0.2 8.2 ± 0.1 (55 ± 5%) 6.6 ± 0.1 6.8 ± 0.2 8.3 ± 0.5
Subsegmental bronchus 6.9 ± 0.2 7.1 ± 0.2 (38 ± 3%) 5.8 ± 0.2 8.6 ± 0.3 (62 ± 5%) 6.8 ± 0.1 6.2 ± 0.8 8.8 ± 0.2
Lung parenchyma 8.5 ± 0.3 (53 ± 4%) 5.9 ± 0.1 7.1 ± 0.2 (37 ± 8%) 5.2 ± 0.2 7.2 ± 0.1 10.2 ± 0.2 (35 ± 2%) 7.1 ± 0.1 9.2 ± 0.2 (66 ± 1%) 6.8 ± 0.2

Competition binding experiments with intact tissue segments were carried out at 4°C. The concentrations of [3H]-NMS and [3H]-CGP-12,177 used were 500 pM.

pKihigh and pKilow: negative logarithm of the equilibrium constants (pKi) at high and low-affinity sites for tested drugs. (%): percentage of high-affinity sites.

Data represent means ± SEM of 4–5 experiments.