Table 1. ACST results on four colorectal cancer datasets.

CC1			CC2			CCms			CCade
Name	pv	adj.pv	Name	pv	adj.pv	Name	pv	adj.pv	Name	pv	adj.p
Colorectal canc	0.001	0.013	Colorectalcanc	0.005	0.095	Colorectalcanc	0.014	0.116	Melanoma	0.005	0.093
MAPK sigpath	0.001	0.013	Cell cycle	0.012	0.106	TLR sig path	0.014	0.116	Insulin sig path	0.013	0.131
TLR sig path	0.002	0.019	Epith cell sig	0.013	0.106	Melanoma	0.015	0.116	Mineral abs	0.019	0.131
Prion diseases	0.003	0.023	Shigellosis	0.014	0.106	Endomet canc	0.018	0.116	Cell cycle	0.020	0.131
Bile secretion	0.005	0.033	Endomet canc	0.019	0.106	Thyroid canc	0.018	0.116	Aldoste-reg sod	0.024	0.131
Epith cell sig	0.008	0.045	Thyroid canc	0.019	0.106	African trypanos	0.020	0.116	Colorectal canc	0.030	0.131
Focal adhesion	0.016	0.078	Wnt sig path	0.023	0.113				Endomet canc	0.032	0.131
Wnt sig path	0.023	0.101	S cell lung canc	0.038	0.166				Thyroid canc	0.032	0.131
			Acute myel leuk	0.050	0.199				Basal cell carc	0.032	0.131
									Hedgehog sigpath	0.036	0.134
									S cell lung cancer	0.048	0.164

The table presents results of ACST on four colorectal cancer datasets. Only significantly altered pathways (adj.p-value<0.25) are presented. Signaling pathways indicated for all datasets are written in bold while signaling pathway indicated more than once are written in italic. Epith cell sig stands for Epithelial cell signaling in Helicobacter pylori infection, S cell lung canc for Small cell lung cancer, African trypanos for African trypanosomiasis, Mineral abs for Mineral absorption and Aldoste-reg sod for Aldosterone-regulated sodium reabsorption.