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. 2012 Mar 28;159(1):156–168. doi: 10.1104/pp.112.194563

Figure 3.

Figure 3.

Stomatal patterning defects conferred by the kob1-3 mutation. DIC images are shown of the abaxial cotyledon epidermis from 17-d-old seedlings of kob1-3 erl1-2 erl2-1 (A), erl1-2 erl2-1 (B), kob1-3 (C), the wild type (wt; D), kob1-3 erecta-105 (E), erecta-105 (F), kob1-3 tmm-1 (G), tmm-1 (H), eld1-1 erl1-2 erl2-1 (I), and eld1-1 (J). While the kob1-3 and eld1-1 mutations do not change stomatal patterning in the wild type (C and J), they lead to the formation of stomata clusters when combined with erl1-2 erl2-1 (A and I). The kob1-3 mutation also promotes excessive asymmetric divisions in the erecta background, leading to the formation of meristemoids (E), and increases the size of stomata clusters in the tmm mutant (G). Images were all taken under the same magnification. Bar = 50 μm.