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. 2012 Aug 7;7(8):e42349. doi: 10.1371/journal.pone.0042349

Figure 4. Genetic interaction of fussel with members of the BMP pathway.

Figure 4

(A) A9-Gal4/y; UAS-saxA. Overexpression of constitutively active sax causes growth of additional vein material between L3–L5 (B) A9-Gal4/y; UAS-saxA; UAS-fussC. Coexpression of fussC ameliorates vein overgrowth. (C) A9-Gal4/y; UAS-mad. Overexpression of mad transforms most of the intervein tissue into vein tissue and eventually results in a blistered wing. (D) A9-Gal4/y; UAS-mad; UAS-fussC. Coexpression of fussC ameliorates blistering and considerably improves vein pattering. (E) A9-Gal4/y; UAS-med. Overexpression of med causes distinct overgrowth and dublication of wing veins. (F) A9-Gal4/y; UAS-med; UAS-fussC. Coexpression of fussC rescues the vein overgrowth and restores the fussC-phenotype.