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. 2012 Apr;165(8):2450–2461. doi: 10.1111/j.1476-5381.2011.01410.x

Figure 7.

Figure 7

Δ8-THCV decreases I/R-induced increased oxidative stress. (A) HNE adducts, a marker for lipid peroxidation/oxidative stress, increase with time following I/R injury. 10 mg·kg−1Δ8-THCV pretreatment attenuates these increases at the time points of reperfusion studied, 6h (I + 6h R) and 24 h (I+24h R). (B) Oxidative modification of proteins, measured by carbonyl adducts, increases with time following I/R injury. 10 mg·kg−1Δ8-THCV pretreatment attenuates these increases at the time points of reperfusion studied, 6h (I + 6h R) and 24 h (I+24h R). Results are mean ± SEM for both panels and n= 8 per group. *P < 0.05 versus vehicle; #P < 0.05 versus corresponding I/R mice.