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. 2012 Jul 5;5(5):636–648. doi: 10.1242/dmm.009266

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© 2012. Published by The Company of Biologists Ltd

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial Share Alike License (http://creativecommons.org/licenses/by-nc-sa/3.0), which permits unrestricted non-commercial use, distribution and reproduction in any medium provided that the original work is properly cited and all further distributions of the work or adaptation are subject to the same Creative Commons License terms.

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Fig. 1. — Incipient kidney renal injury in POKO mice at 4 weeks of age. (A) Representative immunoblot for p27^Kip1 of renal protein from 4-week-old male WT, PPARγ2 KO, ob/ob and POKO mice. Levels were normalized to β-actin. Each value is the optical intensity of each band as a fold induction vs the WT control group (n=6). Fold induction is shown in graphic. *P<0.05 POKO vs ob/ob. (B) Total kidney mRNA levels of different genes from 4-week-old-male mice of each group. Data are means±s.e.m.; n=8–9 in each group; **P<0.01, ***P<0.001 POKO vs ob/ob; ^#P<0.05 POKO vs WT; ^$P<0.05 ob/ob vs WT. Normalized levels with BestKeeper (Bk).