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. 2012 Mar 5;590(Pt 10):2501–2517. doi: 10.1113/jphysiol.2011.225060

Figure 2. The gap junction antagonist MFA blocks oscillations in the AII amacrine–ON cone bipolar cell network.

Figure 2

A, oscillatory activity measured in current clamp from an rd1 ON cone bipolar cell at various times (as indicated on the left) after the onset of an application of 200 μm MFA. Each trace includes the membrane response to a 20 pA depolarizing current pulse lasting for 200 ms. B, the normalized standard deviation of membrane potential recorded from 3 rd1 AII amacrine cells and 7 rd1 ON cone bipolar cells is plotted as a function of time. The data are fitted with a single exponential function.