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. 2012 Aug 16;2(8):801–814. doi: 10.7150/thno.4419

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© Ivyspring International Publisher. This is an open-access article distributed under the terms of the Creative Commons License (http://creativecommons.org/licenses/by-nc-nd/3.0/). Reproduction is permitted for personal, noncommercial use, provided that the article is in whole, unmodified, and properly cited.

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Exploiting cell homing for treating acute myocardial infarction. A) Signaling pathway related to stem cell homing to ischemic tissue. G-CSF promoted cell mobilization and Diprotin A (Dip) inhibited SDF-1 protease, thereby maintaining the stability of cell homing factor SDF-1 released from ischemic tissue. B) G-CSF and Dip dual treatment led to significantly increased myocardial stem cells detected in the ischemic tissue. C) G-CSF and Dip dual treatment increased mouse survival after myocardial infarction. Reproduced with permission from ref 63.