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. Author manuscript; available in PMC: 2013 Aug 1.
Published in final edited form as: Atherosclerosis. 2012 May 31;223(2):365–371. doi: 10.1016/j.atherosclerosis.2012.05.023

Figure 3.

Figure 3

The distributions of plaque phenotypes stratified by remodeling mode in four arteries. The distributions of plaque phenotypes were different between positive and intermediate/negative remodeled lesions in coronary (p<0.001), carotid (p=0.01), renal (p=0.01), and iliac arteries (p=0.04). Compared to intermediate/negative remodeled lesions, positive remodeled lesions demonstrated more characteristics of VH-FA (VH-TCFA and VH-ThCFA) in coronary (84% vs. 25%, p<0.001), carotid (72% vs. 20%, p=0.001), and renal arteries (42% vs. 4%, p=0.001). VH-FA= fibroatheroma; VH-TCFA= thin-capped fibroatheroma; VH-ThCFA= thick-capped fibroatheroma; VH-PIT=pathological intimal thickening; and VH-FC=fibrocalcific plaque.