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. 2012 Aug 20;36(4):255–261. doi: 10.4093/dmj.2012.36.4.255

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Copyright © 2012 Korean Diabetes Association

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 1 — Serum high sensitivity C-reactive protein (hs-CRP) levels and the amounts of urinary 8-hydroxy-2'-deoxyguanosine (8-OHdG) excretion. (A) Comparison between diabetic patients with Gilbert syndrome, GS (+), and those without it, GS (-). Due to their skewed distribution, serum hs-CRP levels (ng/mL) and the amounts of urinary 8-OHdG excretion (urinary 8-OHdG to creatinine ratio, µg/gCr) were log10 transformed before all analyses. (B) Correlation between total bilirubin levels and serum hs-CRP levels. Spearman correlation analysis: r=-0.213, P<0.0001.