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. Author manuscript; available in PMC: 2013 Aug 1.
Published in final edited form as: Neurochem Int. 2012 Jun 16;61(3):389–396. doi: 10.1016/j.neuint.2012.06.003

Figure 2.

Figure 2

Glutamate effect in ATP abundance is not caused by elevated neuronal firing. (A) Blockade of energy synthesis process by KCN lead to a dramatic reduction in ATP levels. In the presence of KCN, glutamate treatment remained to be able to cause ATP reduction. (B) Changes in ATP abundance following KCl-induced neuronal excitation were blocked by TTX; in contrast, blockade of neuronal firing had no effect on glutamate-dependent ATP regulation. *P<0.05, student’s t test.