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Communication between the polarity determinant DivIVA and the cell wall biosynthetic machinery in Streptomyces is bidirectional. The cytoskeletal-like protein DivIVA directs cell wall synthesis at hyphal tips, but when cell wall biosynthetic machinery is compromised, such as when it is inhibited by the antibiotic bacitracin, it communicates back to DivIVA via AfsK-mediated phosphorylation, leading to modulation of apical growth and branching.
