Fig. 13.

Schematic representation of the temporal relationship between all the components of CR(t) as defined by Eq. 11 at F(p) = K. Note that rectangle in the central part is function g₁(t) multiplied by product A₀₁A₀₂Kg₃(φ). It makes most probable crossing of Λ₀ by a random process N(t) near the time at which US occurs. In contrast, the non-associative part of CR(t), connected with earlier portions of the CS is due to crossing of Λ₀ by a process N(t) long before the time at which US appears. It occurs with much lower probability than that of the associative part of CR(t). Pavlov believed that the earlier portion of the CS developed a conditioned inhibition and called it inhibition of delay. Note that the timing of CR(t) is defined by interaction of two major delay sources: the limbic delay time (τ_dl) and the first passage time (τ_fp) of N(t) through threshold Λ₀. The other temporal parameters such as peak time (τ_p). duration (τ_d) and the times of CR onset (τ_on) and offset (τ_off) are only different combinations of these two major delay sources, which allows estimation of variability for each parameter (see Eq. 12) in terms of CVs of these two sources