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. 2012 Sep 13;8(9):e1002688. doi: 10.1371/journal.pcbi.1002688

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This is an open-access article distributed under the terms of the Creative Commons Public Domain declaration, which stipulates that, once placed in the public domain, this work may be freely reproduced, distributed, transmitted, modified, built upon, or otherwise used by anyone for any lawful purpose.

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Shown are the genes predicted to be nonessential under normoxia but essential under hypoxia for the wild type strain. Given the metabolic state shown in Figure 5, the genes predicted to be essential for hypoxic adaptation were mostly located in the glucose/glycerol processing pathways and on the reductive side of the tricarboxylic acid (TCA) cycle. dctA, Na⁺/H⁺-dicarboxylate symporter; eno, enolase; fba, fructose-bisphosphate aldolase; frdA, frdB, frdC, frdD, fumarate reductase; gap, glyceraldehyde-3-phosphate dehydrogenase; gpm, phosphoglycerate mutase; pfKA, pfkB, phosphofructokinase; pgk, phosphoglycerate kinase; ppgK, polyphosphate glucokinase; sdhA, sdhC, sdhD, succinate dehydrogenase; tpi, triosephosphate isomerase.