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Venn diagram of excessively carbonylated proteins throughout the pathogenesis of AD. Enolase, ATP synthase alpha, and UCH-L1 are the common targets of oxidation between AD, MCI, EAD, and PCAD. PCAD, preclinical Alzheimer disease; UCH-L1, ubiquitin carboxy-terminal hydrolase-L1. (To see this illustration in color the reader is referred to the web version of this article at www.liebertonline.com/ars.)
