Table 3.

Effects of Tm2 null alleles on the hdp² phenotype

Genotype	n	Wing position (%)			Muscle fiber phenotype (%)
		U	D	N	H	P	N
hdp2/Y; +/+	25	100	0	0	100	0	0
hdp2/Y; ea5022rxl/+	26	100	0	0	50	50	0
hdp2/Y; DL2/+	12	42	58	0	58	25	17
hdp2/Y; C10/+	36	11	89	0	50	50	0
hdp2/Y; J8/+	26	100	0	0	58	35	7
hdp2/Y; L2/+	22	100	0	0	100	0	0

n = number of flies examined. Wing position: ‘N’ is wild type (normal), ‘U’ is wings raised (up) position typical of hdp², and ‘D’ is wings held alongside (down) the abdomen. Muscle fiber phenotypes were assessed by polarized light microscopy of bisected thoraces. H, hypercontracted' (cf. Figure 1C); P, partially suppressed (see text and Figure 4B); N, wild type/fully suppressed (Figure 1A/1D). Df(3R) ea^5022rxl is a deficiency that completely deletes easter, Tm1 (Erdelyi et al., 1995), and Tm2 (Kreuz et al., 1996); DL2 is a γ-ray induced dominant flightless, recessive lethal mutation of Tm2 associated with a translocation breakpoint within 88F (Sparrow, unpublished) and C10, J8, and L2 are recessive lethal, null Tm2 mutations (Kreuz et al., 1996). All females of genotypes, hdp²/+; Tm2^x/+ (not shown; x = different alleles) hold their wings in the normal position, are flightless, and show a normal IFM pattern.