Notably, different key points of PDGFR signaling can be the object of pharmacological inhibition. First, PDGFRs can be directly blocked by TKIs. Second, the immediate downstream JAK/STAT pathway, SRC kinase and PI3K can be selectively disrupted. Finally, in cases retaining NF-κB activity (around 30% of PTCL-NOSs), different molecules might be targeted. Adapted with permission from Expert Review of Hematology (59).