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Effect of i.p. interleukin (IL)-1β on mouse intestinal permeability. (A) IL-1β caused a concentration-dependent (0–10 mg/kg body weight) increase in mouse intestinal mucosal-to-serosal flux of dextran 10KD. (B) IL-1β caused a time-course (0–120 h) increase in mouse intestinal mucosal-to-serosal flux of dextran 10K. (C) IL-1β caused a time-dependent (0–120 h) drop in mouse intestinal transepithelial resistance (TER) as measured in Ussing chambers. *P<0.001 versus control.
