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. 2012 Aug 9;287(41):34101–34109. doi: 10.1074/jbc.M112.394452

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© 2012 by The American Society for Biochemistry and Molecular Biology, Inc.

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FIGURE 5. — Ac2-26 and mature CRAMP enhance CXCL2 release by CG/NE neutrophils via FPR signaling. A, Ac2-26 and mature CRAMP enhanced the release of CXCL2 from IC-stimulated CG/NE neutrophils in a dose-dependent manner. B, Boc2 inhibited CXCL2 release from IC-stimulated WT neutrophils in a dose-dependent manner. C, fMLF (100 μm) and F2L (100 μm) relieved the inhibition of CXCL2 release by Boc2 (100 μm) in IC-stimulated WT neutrophils. D, fMLF and F2L dose-dependently induced the release of CXCL2 by IC-stimulated CG neutrophils. A–D, CXCL2 levels were determined by ELISA after 2 h of IC stimulation. Values represent mean ± S.E. from at least three independent experiments. *, p < 0.05; **, p < 0.01; ***, p < 0.001 compared with control.